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Predictors of health care utilization in patients with inflammatory bowel disease: a longitudinal study

Publication Year: 1998 Source: Eur J Gastroenterol Hepatol PMID: 9831274 DOI: 10.1097/00042737-199809000-00010

Inflammatory bowel disease (IBD) symptoms may be due not only to the effects of bowel inflammation, but also can result from many non-inflammatory consequences of the disease. Clinical scores and health questionnaire ratings produce a global assessment which is useful in the overall evaluation of the impact of the illness …

Thrombophilia and inflammatory bowel disease: does factor V mutation have a role?

Publication Year: 1998 Source: Eur J Gastroenterol Hepatol PMID: 9831402 DOI: 10.1097/00042737-199810000-00002

BACKGROUND & AIMS: Interleukin (IL)-15 has been found to share many immunoregulatory activities in lymphocytes with IL-2. The aim of this study was to investigate IL-15 activity in organ cultures, localization of IL-15 messenger RNA (mRNA), and proliferation of lamina propria mononuclear cells (LPMCs) in response to recombinant IL-15 using …

Reduced bone mineral density and unbalanced bone metabolism in patients with inflammatory bowel disease

Publication Year: 1998 Source: Inflamm Bowel Dis PMID: 9836078 DOI: 10.1002/ibd.3780040403

INTRODUCTION: Many reports, mainly from the US and Canada but also a recent report from a center in Europe, have documented the increasing impact of Clostridium difficile infections in patients with inflammatory bowel disease (IBD) during the last years. To determine the prevalence of C. difficile infections in hospitalized IBD …

Altered expression of interferon-gamma and interleukin-4 in inflammatory bowel disease

Publication Year: 1998 Source: Inflamm Bowel Dis PMID: 9836081 DOI: 10.1002/ibd.3780040406

An initial genome scan was performed on 540 individuals from 97 families segregating bipolar disorder, collected through the National Institutes of Mental Health Genetics Initiative. We report here affected-sib-pair (ASP) data on 126 marker loci (approximately 68,000 genotypes) mapping to chromosomes 4, 7, 9, 18, 19, 20, and 21q, under …

Inflammatory bowel disease workshop. Vail, Colorado, March 22 and 23, 1998. Maintenance therapy in IBD

Publication Year: 1998 Source: Inflamm Bowel Dis PMID: 9836082 DOI: 10.1002/ibd.3780040408

BACKGROUND: Puberty is thought to be commonly affected in adolescents with inflammatory bowel disease (IBD). AIMS: To determine the impact of Crohn's disease (CD) and ulcerative colitis (UC) on the pubertal growth spurt. METHODS: Retrospective study of 30 boys with CD (CD-M), 11 girls with CD (CD-F), 14 boys with …

Inflammatory bowel disease workshop. Vail, Colorado, March 22 and 23, 1998. Less commonly used therapies for IBD or treatments on the fringe

Publication Year: 1998 Source: Inflamm Bowel Dis PMID: 9836084 DOI: 10.1002/ibd.3780040410

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Complementary and alternative medicine: evaluating its effectiveness in inflammatory bowel disease

Publication Year: 1998 Source: Inflamm Bowel Dis PMID: 9836085 DOI: 10.1002/ibd.3780040411

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The sugar permeability test reflects disease activity in children and adolescents with inflammatory bowel disease

Publication Year: 1998 Source: J Pediatr PMID: 9842038 DOI: 10.1016/s0022-3476(98)70145-3

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Proliferation and apoptosis of lamina propria CD4+ T cells from scid mice with inflammatory bowel disease

Publication Year: 1998 Source: Eur J Immunol PMID: 9842908 DOI: 10.1002/(SICI)1521-4141(199811)28:11<3655::AID-IMMU3655>3.0.CO;2-9

The final composition of leukocytes present in a site of inflammation in response to chemokine stimulation and activation may depend on both the nature of the secreted chemokines as well as the relative expression of the multitude of specific chemokine cell surface receptors on many different cell types. Because related …

Cryptosporidium parvum initiates inflammatory bowel disease in germfree T cell receptor-alpha-deficient mice

Publication Year: 1998 Source: Am J Pathol PMID: 9846962 DOI: 10.1016/s0002-9440(10)65686-6

AIMS: To investigate whether colonic cancer cells exfoliated into the lumen of the organ can be detected by identification of their abnormal CD44 gene products. METHODS: Exfoliated cells were obtained by centrifugation of saline wash-outs of 27 surgically resected colon specimens obtained from 15 patients with carcinoma, seven with ulcerative …

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