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Emerging biotherapies for inflammatory bowel disease
Publication Year: 2003 Source: Expert Opin Emerg Drugs PMID: 14661993 DOI: 10.1517/14728214.8.2.339
A fast, partly recursive deterministic method for calculating Identity-by-Descent (IBD) probabilities was developed with the objective of using IBD in Quantitative Trait Locus (QTL) mapping. The method combined a recursive method for a single marker locus with a method to estimate IBD between sibs using multiple markers. Simulated data was …
Validation of a reliable instrument (QUOTE-IBD) for assessing the quality of health care in Greek patients with inflammatory bowel disease
Publication Year: 2003 Source: Digestion PMID: 14671422 DOI: 10.1159/000075524
BACKGROUND: Indeterminate colitis (IC) remains an enigmatic inflammatory bowel disease (IBD) phenotype. It is currently not clear whether it constitutes merely a problem of terminology, classification, or possibly an early stage of IBD distinct from Crohn's disease (CD) and ulcerative colitis (UC). METHODS: We analysed epidemiological data of studies comparing …
Evaluation of educational programs in inflammatory bowel disease
Publication Year: 2003 Source: Inflamm Bowel Dis PMID: 14671484 DOI: 10.1097/00054725-200311000-00003
The immunopathogenic mechanisms in inflammatory bowel disease (IBD) are not yet fully established. The aim of this study was to determine the profile and magnitude of IgA and IgG autoantibodies in IBD patients. The autoantigen profile defined by IgA and IgG antibodies from 24 IBD (14 Crohn's disease CD], 10 …
Monitoring of AZA/6-MP treatment in children with IBD is necessary
Publication Year: 2003 Source: Inflamm Bowel Dis PMID: 14671488 DOI: 10.1097/00054725-200311000-00007
Not found
Monitoring of azathioprine/6-mercaptopurine treatment in children with IBD is not necessary
Publication Year: 2003 Source: Inflamm Bowel Dis PMID: 14671489 DOI: 10.1097/00054725-200311000-00008
Dysregulated CD4(+) T cell responses and alterations in T regulatory cells (T(reg) cells) play a critical role in autoimmune diseases, including inflammatory bowel disease (IBD). The current study demonstrates that removal of Bcl11b at the double-positive stage of T cell development or only in T(reg) cells causes IBD because of …
Induction of colitis by a CD4+ T cell clone specific for a bacterial epitope
Publication Year: 2003 Source: Proc Natl Acad Sci U S A PMID: 14673119 DOI: 10.1073/pnas.2534546100
Not found
Hold the bone: good news for young women with IBD
Publication Year: 2003 Source: Inflamm Bowel Dis PMID: 14677543 DOI: 10.1097/00054725-200311000-00012
Not found
Increased disease activity in eNOS-deficient mice in experimental colitis
Publication Year: 2003 Source: Free Radic Biol Med PMID: 14680690 DOI: 10.1016/j.freeradbiomed.2003.09.016
Recent reports have focused interest on human cytomegalovirus (HCMV) in inflammatory bowel diseases (IBD). Our aim in this study was to examine the frequency of HCMV-infected intestinal cells in tissue sections obtained from patients with IBD, and to investigate if HCMV-infected intestinal cells produce the proinflammatory cytokine IL-6. We studied …
[Chronic recurrent multifocal osteomyelitis in association with chronic inflammatory bowel disease: entheropathic CRMO]
Publication Year: 2003 Source: Z Rheumatol PMID: 14685714 DOI: 10.1007/s00393-003-0526-7
Glycogen storage disease (GSD)-1b has been associated with neutropenia and abnormalities of neutrophil function. Many individuals with GSD-1b manifest chronic gastrointestinal inflammation. Our study was performed to precisely establish the type, frequency, and spectrum of gastrointestinal disease in patients with GSD-lb. We established a medical database of 36 individuals affected …
Risk of early surgery for Crohn's disease: implications for early treatment strategies
Publication Year: 2003 Source: Am J Gastroenterol PMID: 14687822 DOI: 10.1111/j.1572-0241.2003.08674.x
BACKGROUND: Endogenous opioid peptides have been shown to play a role in the development and/or perpetuation of inflammation. We hypothesize that the endogenous opioid system is involved in inflammatory bowel disease, and antagonism of the opioid-opioid receptor will lead to reversal of inflammation. AIMS: A randomized double-blind placebo-controlled study was …
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